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Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology

ISSN 0893-133X

121 papers in the library · 1,992 citations · publishing 1988-2026

Papers

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Human Ecstasy use is associated with increased cortical excitability: an fMRI study.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology May 2011 Amy L Bauernfeind, Mary S Dietrich, Jennifer U Blackford et al.

The serotonergic neurotoxin, 3,4-methylenedioxymethamphetamine (MDMA/Ecstasy), is a highly popular recreational drug. Human recreational MDMA users have neurocognitive and neuropsychiatric impairments, and human neuroimaging data are consistent with animal reports of serotonin neurotoxicity. However, functional neuroimaging studies have not found consistent effects of MDMA on brain...

Persistent nigrostriatal dopaminergic abnormalities in ex-users of MDMA ('Ecstasy'): an 18F-dopa PET study.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology March 2011 Yen F Tai, Rosa Hoshi, Catherine M Brignell et al.

Ecstasy (±3,4-methylenedioxymethamphetamine, MDMA) is a popular recreational drug with known serotonergic neurotoxicity. Its long-term effects on dopaminergic function are less certain. Studying the long-term effects of ecstasy is often confounded by concomitant polydrug use and the short duration of abstinence. We used (18)F-dopa positron emission tomography (PET) to investigate the long-term...

Chronic adolescent exposure to Δ-9-tetrahydrocannabinol in COMT mutant mice: impact on psychosis-related and other phenotypes.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology October 2010 Colm M P O'Tuathaigh, Magdalena Hryniewiecka, Aine Behan et al.

Cannabis use confers a two-fold increase in the risk for psychosis, with adolescent use conferring even greater risk. A high-low activity catechol-O-methyltransferase (COMT) polymorphism may modulate the effects of adolescent Δ-9-tetrahydrocannabinol (THC) exposure on the risk for adult psychosis. Mice with knockout of the COMT gene were treated chronically with THC (4.0 and 8.0 mg/kg over 20...

A cannabinoid CB(1) receptor antagonist ameliorates impairment of recognition memory on withdrawal from MDMA (Ecstasy).

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology January 2010 Yoko Nawata, Takato Hiranita, Tsuneyuki Yamamoto

(+/-)-3,4-Methylenedioxymethamphetamine (MDMA, 'Ecstasy') abusers have persistent neuropsychiatric deficits including memory impairments after the cessation of abuse. On the other hand, cannabinoid CB(1) receptors have been implicated in learning/memory, and are highly expressed in the hippocampus, a region of the brain believed to have an important function in certain forms of learning and...

Delta 9-tetrahydrocannabinol induces dopamine release in the human striatum.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology February 2009 Matthijs G. Bossong, Bart N M Van Berckel, Ronald Boellaard et al.

The influence of cannabis on mental health receives growing scientific and political attention. An increasing demand for treatment of cannabis dependence has refueled the discussion about the addictive potential of cannabis. A key feature of all addictive drugs is the ability to increase synaptic dopamine levels in the striatum, a mechanism involved in their rewarding and motivating effects....

Chronic delta 9-tetrahydrocannabinol during adolescence provokes sex-dependent changes in the emotional profile in adult rats: behavioral and biochemical correlates.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology October 2008 Tiziana Rubino, Daniela Vigano', Natalia Realini et al.

Few and often contradictory reports exist on the long-term neurobiological consequences of cannabinoid consumption in adolescents. The endocannabinoid system plays an important role during the different stages of brain development as cannabinoids influence the release and action of different neurotransmitters and promote neurogenesis. This study tested whether long-lasting interference by...

Exposure to the selective kappa-opioid receptor agonist salvinorin A modulates the behavioral and molecular effects of cocaine in rats.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology October 2008 Elena H Chartoff, David Potter, Diane Damez-Werno et al.

Stress and chronic exposure to drugs of abuse can trigger addictive and depressive disorders. Both stimuli increase activity of dynorphin, a neuropeptide that acts at kappa-opioid receptors (KORs). In humans, KOR agonists cause dysphoria, raising the possibility that dynorphin modulates the depressive-like effects of stress and chronic drug use. We examined if KOR activation alters sensitivity...

Blunted psychotomimetic and amnestic effects of delta-9-tetrahydrocannabinol in frequent users of cannabis.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology September 2008 Deepak Cyril D'Souza, Mohini Ranganathan, Gabriel Braley et al.

Cannabis is one of the most widely used illicit substances and there is growing interest in the association between cannabis use and psychosis. Delta-9-Tetrahydrocannabinol (Delta-9-THC) the principal active ingredient of cannabis has been shown to induce psychotomimetic and amnestic effects in healthy individuals. Whether people who frequently use cannabis are either protected from or are...

Chronic phenethylamine hallucinogen treatment alters behavioral sensitivity to a metabotropic glutamate 2/3 receptor agonist.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology August 2008 Michael A. Benneyworth, Randy L Smith, Elaine Sanders‐bush

Recent clinical studies in schizophrenic patients show that a selective agonist of group II metabotropic glutamate (mGlu) receptors has robust efficacy in treating positive and negative symptoms. Group II mGlu receptor agonists also modulate the in vivo activity of psychotomimetic drugs, reducing the ability of psychotomimetic hallucinogens to increase glutamatergic transmission. The use of...

Dissociation of the neurochemical and behavioral toxicology of MDMA ('Ecstasy') by citalopram.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology April 2008 Brian J. Piper, Joseph B. Fraiman, Cullen B Owens et al.

High or repeated doses of the recreational drug 3,4-methylenedioxymethamphetamine (MDMA, or 'Ecstasy') produce long-lasting deficits in several markers of serotonin (5-HT) system integrity and also alter behavioral function. However, it is not yet clear whether MDMA-induced serotonergic neurotoxicity is responsible for these behavioral changes or whether other mechanisms are involved. The...

Cannabinoid receptor antagonists counteract sensorimotor gating deficits in the phencyclidine model of psychosis.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology October 1, 2007 Martina Ballmaier, Marco Bortolato, Cristina Rizzetti et al.

Clinical and laboratory findings suggest that cannabinoids and their receptors are implicated in schizophrenia. The role of cannabinoids in schizophrenia remains however poorly understood, as data are often contradictory. The primary aim of this study was to investigate whether the cannabinoid CB1 receptor antagonists rimonabant and AM251 are able to reverse deficits of sensorimotor gating...

Opioid antagonism of cannabinoid effects: differences between marijuana smokers and nonmarijuana smokers.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology June 2007 Margaret Haney

In non-human animals, opioid antagonists block the reinforcing and discriminative-stimulus effects of Delta(9)-tetrahydrocannabinol (THC), while in human marijuana smokers, naltrexone (50 mg) enhances the reinforcing and subjective effects of THC. The objective of this study was to test a lower, more opioid-selective dose of naltrexone (12 mg) in combination with THC. The influence of...

Elevated impulsivity and impaired decision-making in abstinent Ecstasy (MDMA) users compared to polydrug and drug-naïve controls.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology July 2006 Michael John Morgan, Lara Chiara Impallomeni, Alessandro Pirona et al.

Ecstasy (MDMA; 3,4-methylenedioxymethamphetamine) has a well-recognized neurotoxic effect on central serotonergic (5-HT) systems in animals, and there is some evidence of persistent serotonergic dysregulation in human ecstasy users. Serotonin is believed to mediate impulsive behavior and effective decision-making. Thus, the aim of the present study was to investigate impulsive behavior and...

Inhibition of return in the human 5HT2A agonist and NMDA antagonist model of psychosis.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology February 2006 Euphrosyne Gouzoulis‐mayfrank, Karsten Heekeren, Anna Neukirch et al.

Patients with schizophrenia exhibit disturbances of orienting of attention. However, findings have been inconsistent. Pharmacologic challenges with hallucinogens have been used as models for psychosis. The NMDA antagonist state (PCP, ketamine) resembles undifferentiated psychoses with positive and negative symptoms, while the 5-HT(2A) agonist state (LSD, dimethyltryptamine (DMT)) is thought to...

Pharmacokinetic profile of single and repeated oral doses of MDMA in squirrel monkeys: relationship to lasting effects on brain serotonin neurons.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology February 2006 Annis O. Mechan, Jie Yuan, George Hatzidimitriou et al.

A large body of data indicates that (+/-)3,4-methylenedioxymethamphetamine (MDMA, 'ecstasy') can damage brain serotonin neurons in animals. However, the relevance of these preclinical data to humans is uncertain, because doses and routes of administration used in animals have generally differed from those used by humans. Here, we examined the pharmacokinetic profile of MDMA in squirrel monkeys...

Co-administration of THC and MDMA ('ecstasy') synergistically disrupts memory in rats.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology August 2005 June M Young, Iain S. Mcgregor, Paul E Mallet

3,4-Methylenedioxymethamphetamine (MDMA, 'Ecstasy') and cannabis are two of the most commonly used illicit drugs in the western world, and are often used in combination. Very little research has examined their effect on cognitive function or behavior when combined, The present study used a double Y-maze task to examine the acute effect of MDMA and delta9-tetrahydrocannabinol (THC, the principal...

3,4-N-methlenedioxymethamphetamine-induced hypophagia is maintained in 5-HT1B receptor knockout mice, but suppressed by the 5-HT2C receptor antagonist RS102221.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology June 2005 Grégory Conductier, Cyril Crosson, René Hen et al.

3,4-Methylenedioxy-N-methamphetamine (MDMA or 'ecstasy') is a psychoactive substance, first described as an appetite suppressant in humans, inducing side effects and even death. MDMA increases serotonin (5-HT) levels, and 5-HT inhibits food intake, but the 5-HT receptors involved in MDMA-induced changes in feeding behavior are unknown. We examined whether a systemic MDMA injection would reduce...

The psychotomimetic effects of intravenous delta-9-tetrahydrocannabinol in healthy individuals: implications for psychosis.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology August 2004 Deepak Cyril D'Souza, Edward Perry, Lisa Macdougall et al.

Recent advances in the understanding of brain cannabinoid receptor function have renewed interest in the association between cannabinoid compounds and psychosis. In a 3-day, double-blind, randomized, and counterbalanced study, the behavioral, cognitive, and endocrine effects of 0, 2.5, and 5 mg intravenous delta-9-tetrahydrocannabinol (Delta-9-THC) were characterized in 22 healthy individuals,...

Spatial memory deficits induced by perinatal treatment of rats with PCP and reversal effect of D-serine.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology June 1, 2004 Janne Damm Andersen, Bruno Pouzet

It has been suggested that perinatal treatment with the noncompetitive N-methyl-D-aspartate (NMDA) receptor antagonist phencyclidine (PCP) induces transient neurodegeneration in the limbic and cortical structures of rats. Since dysfunction of these structures is associated with cognitive deficits in patients with schizophrenia, we studied the effects of subchronic treatment with PCP in...

Prepulse inhibition and habituation of acoustic startle response in male MDMA ('ecstasy') users, cannabis users, and healthy controls.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology May 2004 Boris B. Quednow, Kai‐uwe Kühn, Klaus Hoenig et al.

Chronic administration of 3,4-methylenedioxymethamphetamine (MDMA) is associated with long-term depletion of serotonin (5-HT) and loss of 5-HT axons in the brains of rodents and nonhuman primates. Despite the broad database concerning the selective serotonergic neurotoxicity of recreational MDMA consumption by humans, controversy still exists with respect to the question of whether the...

Effects of THC on behavioral measures of impulsivity in humans.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology July 2003 Jennifer McDonald, Laura Schleifer, Jerry B Richards et al.

This study investigated the acute effects of delta(9)-tetrahydrocannabinol (THC) on four behavioral measures of impulsivity in recreational marijuana users. Although impulsive behavior has been studied using several different measures of impulsivity, few studies have utilized more than one of these measures on a single cohort. In this study, 37 healthy men and women participated in three...

Induction of metabolic hypofunction and neurochemical deficits after chronic intermittent exposure to phencyclidine: differential modulation by antipsychotic drugs.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology February 1, 2003 Susan M Cochran, Matthew D Kennedy, Clare E McKerchar et al.

Numerous human imaging studies have revealed an absolute or relative metabolic hypofunction within the prefrontal cortex, thalamus and temporal lobes of schizophrenic patients. The former deficit correlates with cognitive deficits and negative symptoms, whereas the latter correlates with positive symptomologies. There is also general consensus that schizophrenia is associated with decreased...

Subchronic continuous phencyclidine administration potentiates amphetamine-induced frontal cortex dopamine release.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology 2003 Andrea Balla, Henry Sershen, Michael Serra et al.

Functional dopaminergic hyperactivity is a key feature of schizophrenia. Etiology of this dopaminergic hyperactivity, however, is unknown. We have recently demonstrated that subchronic phencyclidine (PCP) treatment in rodents induces striatal dopaminergic hyperactivity similar to that observed in schizophrenia. The present study investigates the ability of PCP to potentiate amphetamine-induced...

Effects of acute smoked marijuana on complex cognitive performance.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology November 2001 Carl L. Hart, W Van Gorp, M Haney et al.

Although the ability to perform complex cognitive operations is assumed to be impaired following acute marijuana smoking, complex cognitive performance after acute marijuana use has not been adequately assessed under experimental conditions. In the present study, we used a within-participant double-blind design to evaluate the effects acute marijuana smoking on complex cognitive performance in...

Effects of ketamine in normal and schizophrenic volunteers.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology October 1, 2001 Adrienne C. Lahti, M A Weiler, B A Tamara Michaelidis et al.

This study evaluates the effects of ketamine on healthy and schizophrenic volunteers (SVs) in an effort to define the detailed behavioral effects of the drug in a psychosis model. We compared the effects of ketamine on normal and SVs to establish the comparability of their responses and the extent to which normal subjects might be used experimentally as a model. Eighteen normal volunteers (NVs)...