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Marco Gesi

4 papers in the library · 78 citations · publishing 2001-2011

Papers

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MDMA (ecstasy) enhances loud noise-induced morphofunctional alterations in heart and adrenal gland.

Microscopy research and technique September 2011 Federica Fulceri, Michela Ferrucci, Paola Lenzi et al.

Noise is an environmental stressor increasingly more present in modern life and, in particular, in a variety of recreational contexts. The aim of this work is to show the effects of noise on the myocardium and adrenal gland, through a careful review of the literature dealing with the peripheral effects of noise exposure in experimental and clinical studies. Noise induces adverse effects in...

MDMA Induces Caspase‐3 Activation in the Limbic System but not in Striatum

Annals of the New York Academy of Sciences August 1, 2006 Ilaria Tamburini, Fabio Blandini, Marco Gesi et al. 24 citations

Abstract: Several studies, carried out in chronic (+/−) 3,4‐methylenedioxymethamphetamine (MDMA) abusers, have shown memory loss and cognitive impairment, as well as persistent electroencephalographic changes. This suggests that, at least in humans, forebrain areas, including the limbic system, might be altered by MDMA. Consistently, recent experimental evidences suggest that, in rodents, MDMA,...

Loud noise enhances nigrostriatal dopamine toxicity induced by MDMA in mice.

Microscopy research and technique July 1, 2004 Marco Gesi, Michela Ferrucci, Mario Giusiani et al.

The neurotoxicity of 3,4-methylenedioxymethamphetamine (MDMA, ecstasy) has been intensely investigated due to the widespread abuse of this drug and its neurotoxic effects. In mice, MDMA neurotoxicity has been demonstrated for striatal dopamine (DA) terminals. However, the current literature has reported great variability in the effects induced by MDMA; this is partially due to changes in...

Biochemical effects of the monoamine neurotoxins DSP-4 and MDMA in specific brain regions of MAO-B-deficient mice

Synapse 2001 Francesco Fornai, Filippo Sean Giorgi, Marco Gesi et al. 54 citations

Previous studies reported that drugs acting as monoamine oxidase (MAO)-B inhibitors prevented biochemical effects induced by the neurotoxins N-(2-chloroethyl)-N-ethyl-2-bromobenzylamine (DSP-4) and 3,4-methylenedioxymethamphetamine (MDMA, "ecstasy"). In this study, we administered DSP-4 (50 mg/kg) or MDMA (50 mg/kg x 2, 2 h apart) to MAO-B deficient mice. Monoamine content in various brain...