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Eric A. Miller

4 papers in the library · 16 citations · publishing 2023-2025

Papers

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Psychedelics for Alcohol Use Disorder: A Narrative Review with Candidate Mechanisms of Action.

CNS Drugs July 10, 2025 Eric A. Miller, Christy Capone, Erica Eaton et al. 4 citations

Psychedelics have been studied since the 1950s as a potential treatment for alcohol use disorder (AUD), with over a dozen clinical trials of lysergic acid diethylamide (LSD), and several contemporary trials of psilocybin and ayahuasca for this indication. Herein, we characterize foundational studies from the 1950s to the present, with emphasis on key design factors that varied considerably...

Predicting non-response to ketamine for depression: An exploratory symptom-level analysis of real-world data among military veterans.

Psychiatry Research May 1, 2024 Eric A. Miller, Houtan Totonchi Afshar, Jyoti Mishra et al. 12 citations

Ketamine helps some patients with treatment resistant depression (TRD), but reliable methods for predicting which patients will, or will not, respond to treatment are lacking. Herein, we aim to inform prediction models of non-response to ketamine/esketamine in adults with TRD. This is a retrospective analysis of PHQ-9 item response data from 120 patients with TRD who received repeated doses of...

Predicting non-response to ketamine for depression: a symptom-level analysis of real-world data

medRxiv Preprint Server July 3, 2023 Eric A. Miller, Houtan Totonchi Afshar, Jyoti Mishra et al. preprint

Background Ketamine helps some patients with treatment resistant depression (TRD), but reliable methods for predicting which patients will, or will not, respond to treatment are lacking.

Effects of cannabis use on antidepressant treatment response to repetitive transcranial magnetic stimulation and ketamine

medRxiv Preprint Server June 28, 2023 Mohammad Ali Shenasa, Houtan Totonchi Afshar, Eric A. Miller et al. preprint

Background The antidepressant effects of ketamine and repetitive transcranial magnetic stimulation (rTMS) are hypothesized to rely on mechanisms of long-term-potentiation and synaptic plasticity. Cannabis, via activation of CB1 receptors has been shown to impair synaptic plasticity, suggesting that cannabis use might moderate the antidepressant effects of ketamine and/or rTMS.