How Can Consciousness Propagate Through Chemical Synapses? Conscious Ontogenetic Development Through Phasic Release and Synaptic Density, Within Subconscious Priors of Tonic Stimulation

Abstract

The transmission of consciousness through a chemical synapses is an under-specified probable occurrence. Chemical transmission is essential for neuronal propagation and neural propagation is essential within many existing measurements of consciousness. The development and plasticity of consciousness throughout an animal's lifetime requires grounded chemical distinctions, executive and non-executive processing and discrete neurotransmission should be neurobiologically consistent with existing accounts of consciousness. These hypotheses propose chemical-conscious correlates between neurons and are attached through evolution to established cognitive and environmental mechanisms. These hypotheses suggest consciousness may be chemically affected at global scales and developed ontogenetically during continuous environmental rationalization and all subsequent propagation. Single-synaptic phasic transmitter transients are positioned as executive signals within neural propagation, selecting synaptic and possibly epigenetic reconsolidation while directing attention towards significance within tonic stimulation. This framing is consistent with phasic and tonic dynamics, with each transmitter being differentiated by their established functions. Phasic transients are further suggested to direct cortical spreading (an established signature of consciousness) under tonic, epigenetic, and synaptic allowance. Because phasic release fundamentally affects synaptic plasticity, while synapses determine the threshold and magnitude of phasic response, this framing allows a developmental and individually variable model of consciousness. Existing theories may be extended under this hypothetical developmental framing, such as Predictive Processing, Global Neuronal Workspace Theory (GNWT), Integrated Information Theory (IIT), and Affective Neuroscience; extending each framework by approaching phasic response as a significance signal at the synaptic scale, providing mechanisms through which present conscious experience updates future conscious experience. Because each hypothesis is falsifiable, provided predictions and experimental directions can largely determine the accuracy of each claim; given hypotheses are precise enough to fail and many of their correlating experiments are practical enough to proceed. If this account holds, it points toward practical improvements in consciousness research, chemical synaptic mechanisms, depression treatments, therapeutic approaches, memory rehabilitation, learning environment refinement strategies, and early developmental interventions.