The Influence of Faradarmani Consciousness Field on the Survival and Death of MCF-7 Breast Cancer Cells: An Optimization Perspective
Mohammad Ali Taheri, Majid Mahdavi, Zohreh Afsartala, Laleh Amani, Farid Semsarha
The Scientific Journal of Cosmointel April 17, 2022 DOI: 10.61450/joci.v1i6.53 (opens in new tab)
Study at a glance
AI-extracted from the abstract| Characteristics | Laboratory experimental study Peer reviewed |
|---|---|
| Population | MCF-7 breast cancer cell line in culture |
| Intervention | Faradarmani Consciousness Field |
| Duration | 6–24 hours of treatment; cell cycle and gene expression assessed up to 24 hours |
| Measures | MTT assay, cell cycle analysis, real-time RT-PCR for Bcl2 and Bax |
| Citations | 28 |
| Key findings | The authors report that the Faradarmani Consciousness Field increased MCF-7 cell viability by about 18% versus control, raised S-phase cells by 56% up to 24 hours, and decreased the Bax/Bcl2 ratio after 24 hours, which they interpret as enhanced survival and resistance to death. They argue these results are consistent with Taheri's claim that proliferation and survival become optimized under the field. |
Abstract
Mohammad Ali Taheri, the founder of the Faradarmani Consciousness Field (CF) maintains that beyond matter and energy, there are Taheri Consciousness Fields (TCFs) that can be accessed through the connection of the consciousness of the subject of study to the Cosmic Consciousness Network (CCN). This causes an optimization in a subject’s structure and function. Apart from the fact that most studies in the field of cancer are aimed at the death of cancer cells, the purpose of this study was to investigate the MCF-7 cancer cell survival and death according to their optimal nature under the influence of the Faradarmani CF. For the purpose of this investigation, cellular viability was evaluated by MTT assay for 6-24 h under the influence of the Faradarmani CF. Consequently, cell cycle analysis was performed for the evaluation of apoptotic cell death. Finally, the expression of Bcl2 and Bax genes in MCF-7 cells was evaluated as a validation of the obtained cellular scale results. Our findings demonstrate that the Faradarmani CF treatment significantly induced cell proliferation in the MCF-7 cell culture in specific test conditions mediated by increasing cellular viability by about 18% in comparison to the control in a time-dependent manner. Moreover, the S-phase in the cell cycle, as a sign of cell population in the sample treated with the Faradarmani CF, was increased by 56% (up to 24h) in comparison to the control. The real-time RT-PCR reaction results show that in cells treated with the Faradarmani CF, the Bax/Bcl2 ratio decreased after 24 h (more than the 1-fold negative control (untreated)), suggesting a higher cell survival and resistance to death. Considering the concepts of the TCFs, according to Taheri, proliferation and survival appear to become optimized in MCF-7 cancer cell lines under the influence of the Faradarmani CF.
Citations in the library
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and 2 more in the library