Ingestion of Synthetic Street Drug “25-I” (25I-NBOMe) Causing Type B Lactic Acidosis and Multi-Organ Dysfunction
M. Friedman, Marcelo R. Malakooti, Craig M. Smith, Z. Harris, M. Wainwright
February 27, 2015 DOI: 10.14740/jmc.v6i3.2023 (opens in new tab)
Study at a glance
AI-extracted from the abstract| Characteristics | Case report |
|---|---|
| Sample size | 1 |
| Population | A 16-year-old male who ingested 25I-NBOMe |
| Interventions | Thiamine L-carnitine coenzyme Q-10 n-acetylcysteine plasmapheresis |
| Key findings | The patient developed type B lactic acidosis peaking at 8.5 mEq/L 38 hours after 25-I ingestion, with elevated mixed venous saturation and decreased arterial-venous oxygen content difference. Treatment with thiamine, L-carnitine, coenzyme Q-10, n-acetylcysteine, and plasmapheresis reduced lactate and organ injury markers, normalized the oxygen content difference, and led to full recovery. |
Abstract
25I-NBOMe (25-I) is a novel synthetic agonist of the 5-HT2A receptor. It is used recreationally for its psychedelic properties similar to LSD. Side effects include altered mental status, sympathomimetic symptoms, serotonin syndrome and multiple organ injury. We report a case of delayed type B lactic acidosis after ingestion of 25-I. A 16-year-old male presented after being found obtunded. He developed seizures requiring endotracheal intubation. He developed a lactic acidosis about 30 hours after ingestion, peaking at 8.5 mEq/L 38 hours post-ingestion. Concurrently the patient had an elevated mixed venous saturation and decreased arterial-venous oxygen content difference, indicating a type B lactic acidosis. Thiamine, L-carnitine, and coenzyme Q-10 were initiated to treat the metabolic derangement and promote oxygen utilization. Additional treatments included n-acetylcysteine and plasmapheresis. This treatment strategy resulted in reduction in lactate and other markers of organ injury. Arterial-venous oxygen content difference increased to normal levels. The patient made a full recovery. Type B lactic acidosis has been reported previously in a variety of diseases, but never after 25-I ingestion. A novel approach of vitamins and co-factors to support mitochondrial function, n-acetylcysteine and plasmapheresis was employed to treat the lactic acidosis and multi-organ dysfunction successfully. J Med Cases. 2015;6(3):125-127 doi: http://dx.doi.org/10.14740/jmc2023w
Citations in the library
Cites 3
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