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Towards an expanded neurocognitive account of ketamine's rapid antidepressant effects.

Yingliang Dai, Ben J. Harrison, Christopher G. Davey, Trevor Steward

The International Journal of Neuropsychopharmacology February 4, 2025 DOI: 10.1093/ijnp/pyaf010 (opens in new tab)

Study at a glance

AI-extracted from the abstract
Characteristics Review Peer reviewed
Intervention Ketamine
Topics Depression Ketamine Esketamine
Keywords Neurocognitive Depression/mental-health/mood-disorders Ketamine/psychopharmacology/antidepressants Neuroscience/neurobiology/brain-research
Citations 6
Key findings Ketamine's antidepressant effects are driven by modulation of brain systems supporting reward processing, interoception, and self-related cognition.

Abstract

Ketamine is an N-methyl-D-aspartate receptor antagonist that has shown effectiveness as a rapidly acting treatment for depression. Although advances have been made in understanding ketamine's antidepressant pharmacological and molecular mechanisms of action, the large-scale neurocognitive mechanisms driving its therapeutic effects are less clearly understood. To help provide such a framework, we provide a synthesis of current evidence linking ketamine treatment to the modulation of brain systems supporting reward processing, interoception, and self-related cognition. We suggest that ketamine's antidepressant effects are, at least in part, driven by dynamic multi-level influences across these key functional domains.

Comparable studies

Other narrative reviews on ketamine for depression, most cited first.

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