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Marina R. Picciotto

4 papers in the library · 475 citations · publishing 2019-2025

Papers

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Effects of ketamine on GABAergic and glutamatergic activity in the mPFC: biphasic recruitment of GABA function in antidepressant-like responses.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology March 1, 2025 Manoela V. Fogaça, Fernanda Daher, Marina R. Picciotto 22 citations

Major depressive disorder (MDD) is associated with disruptions in glutamatergic and GABAergic activity in the medial prefrontal cortex (mPFC), leading to altered synaptic formation and function. Low doses of ketamine rapidly rescue these deficits, inducing fast and sustained antidepressant effects. While it is suggested that ketamine produces a rapid glutamatergic enhancement in the mPFC, the...

Slow and fast cortical cholinergic arousal is reduced in a mouse model of focal seizures with impaired consciousness.

Cell Reports December 24, 2024 Lim-Anna Sieu, Shobhit Singla, Jiayang Liu et al. 8 citations

Patients with focal temporal lobe seizures often experience loss of consciousness associated with cortical slow waves, like those in deep sleep. Previous work in rat models suggests that decreased subcortical arousal causes depressed cortical function during focal seizures. However, these studies were performed under light anesthesia, making it impossible to correlate conscious behavior with...

GABA interneurons are the cellular trigger for ketamine's rapid antidepressant actions.

Journal of Clinical Investigation November 19, 2019 Danielle M. Gerhard, Santosh Pothula, Rong-Jian Liu et al. 345 citations

A single sub-anesthetic dose of ketamine, an NMDA receptor (NMDAR) antagonist, produces rapid and sustained antidepressant actions in depressed patients, addressing a major unmet need for the treatment of mood disorders. Ketamine produces a rapid increase in extracellular glutamate and synaptic formation in the prefrontal cortex, but the initial cellular trigger that initiates these and its...

Role of Neuronal VEGF Signaling in the Prefrontal Cortex in the Rapid Antidepressant Effects of Ketamine

American Journal of Psychiatry January 4, 2019 Satoshi Deyama, Eunyoung Bang, Eric S. Wohleb et al. 100 citations

OBJECTIVE: -methyl-d-aspartate receptor antagonist ketamine produces rapid and sustained antidepressant actions even in patients with treatment-resistant depression. Vascular endothelial growth factor (VEGF) has been implicated in the effects of conventional monoamine-based antidepressants, but the role of VEGF in the rapid antidepressant actions of ketamine remains unclear. The authors...