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Safety of ibogaine administration in detoxification of opioid-dependent individuals: a descriptive open-label observational study.

Thomas Knuijver, Arnt Schellekens, Maarten Belgers, Rogier Donders, Toon Van Oosteren, Kees Kramers, Robbert J. Verkes

Addiction (Abingdon, England) 2022 DOI: 10.1111/add.15448 (opens in new tab)

Study at a glance

AI-extracted from the abstract
Characteristics Open-label observational study Peer reviewed
Sample size 14
Population Patients with opioid use disorder on opioid maintenance treatment who failed to reach abstinence with standard care
Intervention Ibogaine-HCl
Dose 10 mg/kg
Duration At least 24 hours
Topics Addiction Ibogaine
Keywords Cardiac safety Cerebellar toxicity Detoxification Ibogaine: ibogaine Ataxia Withdrawal effects Psychomimetic effects Significant qtc prolongation
Citations 49
Key findings Ibogaine treatment induced clinically relevant but reversible QTc prolongation, bradycardia, and severe ataxia in patients with opioid use disorder.

Abstract

Ibogaine is an indole alkaloid used in rituals of the African Bwiti tribe. It is also used in non-medical settings to treat addiction. However, ibogaine has been linked to several deaths, mainly due to cardiac events called torsades des pointes preceded by QTc prolongation as well as other safety concerns. This study aimed to evaluate the cardiac, cerebellar and psychomimetic safety of ibogaine in patients with opioid use disorder. A descriptive open-label observational study. Department of psychiatry in a university medical center, the Netherlands. Patients with opioid use disorder (n = 14) on opioid maintenance treatment with a lasting wish for abstinence, who failed to reach abstinence with standard care. After conversion to morphine-sulphate, a single dose of ibogaine-HCl 10 mg/kg was administered and patients were monitored at regular intervals for at least 24 hours assessing QTc, blood pressure and heart rate, scale for the assessment and rating of ataxia (SARA) to assess cerebellar side effects and the delirium observation scale (DOS) to assess psychomimetic effects. The maximum QTc (Fridericia) prolongation was on average 95ms (range 29-146ms). Fifty percent of subjects reached a QTc of over 500ms during the observation period. In six out 14 subjects prolongation above 450ms lasted beyond 24 hours after ingestion of ibogaine. No torsades des pointes were observed. Severe transient ataxia with inability to walk without support was seen in all patients. Withdrawal and psychomimetic effects were mostly well-tolerated and manageable (11/14 did not return to morphine within 24 hours, DOS scores remained below threshold). This open-label observational study found that ibogaine treatment of patients with opioid use disorder can induce a clinically relevant but reversible QTc prolongation, bradycardia, and severe ataxia.

Comparable studies

Other non-randomized and open-label trials on ibogaine for addiction, most cited first.

Study Year Design Participants
Medication Development of Ibogaine as a Pharmacotherapy for Drug Dependencea. Cocaine-dependent patients 1998 Rising tolerance study (Phase I trial)
Ibogaine Detoxification Transitions Opioid and Cocaine Abusers Between Dependence and Abstinence: Clinical Observations and Treatment Outcomes. Human volunteers seeking to detoxify from opioids or cocaine 2018 Open-label case series n = 191
Feasibility, safety and preliminary effects of ibogaine in patients with moderate and severe alcohol use disorder: a pilot, open-label study Adults with moderate to severe alcohol use disorder 2026 Open-label pilot feasibility study n = 9
Hallucinogen Persisting Perception Disorder After Ibogaine Treatment for Opioid Dependence. One healthy adult volunteer 2018 Case study (open-label trial, single subject) n = 1

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