Skip to content

Research Progress on the Mechanism of Ketamine in Neuropathic Pain Comorbid Depression

Qing-Mei Li, Shuming Liu, Bangyong Qin, Xing-Feng Liu

Neuropsychiatric Disease and Treatment August 9, 2025 DOI: 10.2147/ndt.s534938 (opens in new tab)

Study at a glance

AI-extracted from the abstract
Characteristics Review Peer reviewed
Intervention Ketamine
Topics Depression Esketamine Ketamine
Key findings The review proposes that ketamine's antidepressant action involves disinhibition and neuroplasticity mechanisms and specific brain regions such as the hippocampus and anterior cingulate cortex, and argues that integrating bioinformatics and molecular biology can clarify its therapeutic effects and mechanisms in neuropathic pain comorbid with depression.

Abstract

Abstract This review systematically summarizes the research progress of ketamine and its interaction with specific brain regions in the context of neuropathic pain comorbid with depression. As a non-competitive inhibitor of N-methyl-D-aspartate (NMDA) receptors, Ketamine exerts complex mechanisms and controversial topic. The structure and function of NMDA receptors, as well as the binding sites of ketamine, are summarized, and the hypothesis of disinhibition and neuroplasticity of ketamine’s antidepressant effect is elaborated. The mechanism of key brain regions, such as hippocampus and anterior cingulate cortex, is discussed in detail, and the antidepressant effect of ketamine is explored from the perspective of transcriptomics. Finally, this review integrates bioinformatics, molecular biology and other interdisciplinary approaches to elucidate the therapeutic effects and potential mechanisms of ketamine in treating neuropathic pain and depression comorbidity, providing a comprehensive theoretical basis, new directions for subsequent research and novel insights for the clinical application of ketamine.

Comparable studies

Other narrative reviews on ketamine for depression, most cited first.

Explore topics

By condition and practice