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January 2026

Default mode network

What January 2026's 6 new studies found, synthesized from the papers below. All Default mode network research →

The synthesis

Synthesized from 6 studies in the library · AI-generated, grounded in the abstracts below

Found by searching the library for Default mode network, DMN, resting-state network, then ranked by relevance.

Research in January 2026 consistently identifies the default mode network (DMN) as a key neural substrate in psychedelic therapy, hypnosis, and psychiatric disorders, with findings showing that psychedelics like psilocybin reduce DMN activity and rigidity, correlating with therapeutic benefits. However, the evidence is largely from reviews and theoretical frameworks rather than new empirical studies, and one study on multiple sclerosis suggests DMN disruption may also occur in non-psychedelic contexts, indicating the DMN's role is complex and not fully understood.

Evidence by study

Direction is which way each study's own result points, not our rating of the study.

What the directions mean
Supports:
the study found the intervention worked, or its hypothesis held.
Opposes:
it found the opposite, no benefit or a harm.
No effect:
no significant difference either way.
Mixed:
effects in both directions within the same study.
Unclear:
the abstract does not report a direction.

Hypnosis modulates the default mode network (DMN) along with other networks to reorganize emotional experience and self-referential processing.

review

Psilocybin reduces DMN activity and connectivity, contributing to rapid and sustained antidepressant effects.

review

Psilocybin and LSD cause disintegration of the DMN, which correlates with alleviation of rigid cognitive patterns in depression and anxiety.

systematic review

Altered excitation-inhibition balance is observed in the DMN in multiple sclerosis, but the direction of effect on DMN function is not specified.

observational

The reflective self, underpinned by DMN activity, is contrasted with an experiential self state, and altered states like meditation can shift away from DMN-dominated processing.

theoretical

OCD is associated with a hyperfunctioning DMN, and psilocybin may break rigid neuronal patterns by modulating the DMN.

review

Points of agreement

  • Psychedelics (psilocybin, LSD) reduce DMN activity and connectivity, which is linked to therapeutic effects in depression, anxiety, and OCD.
  • Hypnosis and meditation also modulate the DMN, suggesting a common pathway for altering self-referential processing.
  • DMN dysfunction is implicated in multiple psychiatric and neurological conditions, including OCD and multiple sclerosis.

Conflicts

  • One study on multiple sclerosis reports altered DMN excitation-inhibition balance, but the direction (whether increased or decreased DMN activity) is unclear, contrasting with the consistent reduction seen in psychedelic studies.
  • The theoretical framework in one study distinguishes between reflective (DMN-based) and experiential self states, but other studies do not explicitly adopt this dual-self model.

Gaps

  • No new empirical neuroimaging data from January 2026 are provided; all studies are reviews or theoretical.
  • The durability of DMN changes beyond the acute psychedelic experience is not addressed in the provided abstracts.
  • Sample sizes and specific populations are not reported for most studies, limiting generalizability.
  • The relationship between DMN modulation and clinical outcomes is correlational, not causal, in the reviewed evidence.
Browse these studies in the library