Some Chemical Mechanisms Involved in the Formation of Anomalous Pigmented Birthmarks in Cases of the Reincarnation Type
Journal of Scientific Exploration October 15, 2025 DOI: 10.31275/20253455 (opens in new tab)
Study at a glance
AI-extracted from the abstract| Characteristics | Theoretical or philosophical paper Peer reviewed |
|---|---|
| Key points | Argues that micro-PK could influence the formation of anomalous pigmented birthmarks by acting on probabilistic quantum events that alter the motion of migrating melanocyte and melanocyte precursor cells, and proposes two experiments to test effects of consciousness on this cell migration. |
Abstract
In Ian Stevenson’s time, little was known about the formation of pigmented birthmarks. Today, this is no longer true due in part to advances made to understand better and combat melanoma, an aggressive form of skin cancer that forms from melanocyte cells (which give the skin pigment and form pigmented birthmarks). Here, we summarize what is now known about the circumstances surrounding the formation of pigmented birthmarks during fetal development and some relevant work on micro-PK and its role in biology. We then enumerate some chemical mechanisms that are likely involved in the formation of anomalous pigmented birthmarks in cases of the reincarnation type. These mechanisms include the radical-pair mechanism, which has been the focus of much attention in quantum biology, as well as potential quantum computations occurring in various cytoskeletal filaments. All of the mechanisms enumerated here potentially alter the motion of migrating melanocyte and melanocyte precursor cells during pigmented birthmark formation based on the results of many simultaneous probabilistic quantum mechanical events. We theorize that micro-PK should be able to act on these probabilistic quantum events and thus exert influence on the motion of the migrating cells. We propose two experiments to further investigate the effects of consciousness on the motion of migrating melanocyte and melanocyte precursor cells.