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Behavioral effects of (+-) 3,4-methylenedioxyamphetamine (MDA) and (+-) 3,4-methylenedioxymethamphetamine (MDMA) in the pigeon: interactions with noradrenergic and serotonergic systems.

M A Nader, S M Hoffmann, J E Barrett

Psychopharmacology 1989 DOI: 10.1007/bf00444689 (opens in new tab)

Study at a glance

AI-extracted from the abstract
Characteristics Behavioral pharmacology experiment Peer reviewed
Population Pigeons key pecking under a multiple fixed-interval, fixed-ratio schedule of food presentation
Interventions MDA MDMA MDE Metergoline Ketanserin Prazosin
Dose MDA, MDMA, and MDE: 0.1-10.0 mg/kg; Metergoline: 0.1-1.0 mg/kg; Ketanserin: 0.1-3.0 mg/kg; Prazosin: 0.3-3.0 mg/kg
Topics MDMA Serotonin
Key findings MDA, MDMA, and MDE either had no effect or decreased response rates dose-dependently, with MDA at least 1 log unit more potent. Metergoline and ketanserin blocked MDA's rate-decreasing effects but not MDMA's, while prazosin blocked MDMA's effects but not MDA's. The authors conclude that MDA and MDMA, though structurally similar, appear to act through different neurotransmitter systems.

Abstract

The effects of three amphetamine analogs were assessed in pigeons key pecking under a multiple 3-min fixed-interval (FI), 30 response fixed-ratio (FR) schedule of food presentation. At doses between 0.1 and 10.0 mg/kg, (+-) 3,4-methylenedioxyamphetamine (MDA), (+-) 3,4-methylenedioxymethamphetamine (MDMA), and (+-)-N-ethyl-3,4-methylenedioxyamphetamine (MDE) either had no effect or decreased response rates in both components of the multiple schedule in a dose-dependent manner. MDA was at least 1 log unit more potent than the other two compounds. Metergoline (0.1-1.0 mg/kg), a serotonin (5-HT) antagonist with comparable affinity for the 5-HT1 and 5-HT2 receptor subtypes, blocked the rate-decreasing effects of 3.0 mg/kg MDA in both components of the multiple schedule, but did not affect the MDMA dose-response curve. The 5-HT2 receptor antagonist ketanserin (0.1-3.0 mg/kg) also restored FI and FR responding that was decreased by 3.0 mg/kg MDA, but had no effect on responding suppressed by MDMA. The noradrenergic alpha-1 antagonist prazosin (0.3-3.0 mg/kg) blocked the behavioral effects of 3.0 mg/kg MDMA at doses that did not attenuate MDA's rate-decreasing effects. These results indicate that although MDA and MDMA are structurally similar and have similar behavioral effects, their actions appear to be mediated through different neurotransmitter systems.