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DXM, CYP2D6-inhibiting antidepressants, piracetam, and glutamine: proposing a ketamine-class antidepressant regimen with existing drugs.

Ngo Cheung

Frontiers in Psychiatry 2026 DOI: 10.3389/fpsyt.2026.1751605 (opens in new tab)

Study at a glance

AI-extracted from the abstract
Characteristics Theoretical or philosophical paper Peer reviewed
Interventions dextromethorphan CYP2D6 inhibitor piracetam L-glutamine
Topics Depression Ketamine Esketamine
Keywords Cyp2d6-inhibiting antidepressants Dxm Glutamatergic Glutamine Piracetam
Citations 6
Key points Proposes that a four-component oral regimen may approximate ketamine's full plasticity cascade for rapid antidepressant effects, but remains untested in humans.

Abstract

Rapid-acting antidepressants show that mood can lift within hours when glutamatergic circuits shift from an "NMDA-dominant" to an "AMPA-dominant" state. Intravenous ketamine achieves this flip but is hampered by dissociation and logistics, while dextromethorphan + bupropion (Auvelity®) primarily supplies the initial NMDA blockade and yields slower, less durable benefit. We hypothesize that a fully oral, low-cost, four-component regimen may be able to approximate ketamine's full plasticity cascade (1) dextromethorphan (DXM) for NMDA antagonism; (2) a potent CYP2D6 inhibitor (fluoxetine, paroxetine, or high-dose duloxetine) to prolong DXM exposure; (3) piracetam as an AMPA positive allosteric modulator; and (4) micronized L-glutamine to restore presynaptic glutamate pools and buffer against excitotoxicity. Preclinical evidence supports mechanistic synergy along the same axis, but the full combination remains untested in humans. This hypothesis warrants formal preclinical and clinical evaluation.

Comparable studies

Other theoretical and historical papers on ketamine for depression, most cited first.

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