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Breaking through anhedonia: How ketamine reignites the drive for rewards.

Colleen A McClung

Neuron May 7, 2025 DOI: 10.1016/j.neuron.2025.03.025 (opens in new tab)

Study at a glance

AI-extracted from the abstract
Characteristics Experimental study Peer reviewed
Population Nucleus accumbens D1 dopamine receptor-expressing medium spiny neurons (D1-MSNs) in a stress model
Intervention Ketamine
Topics Esketamine Ketamine
Keywords Mental health Psychopharmacology Addiction treatment Brain chemistry
Key findings Ketamine reverses stress-induced changes in excitatory synapses in nucleus accumbens D1-MSNs, and these changes are necessary for the treatment of anhedonia-like behavior.

Abstract

In this issue of Neuron, Pignatelli et al.1 find that ketamine reverses stress-induced changes in excitatory synapses in nucleus accumbens D1 dopamine receptor-expressing medium spiny neurons (D1-MSNs) and that these changes are necessary for the treatment of anhedonia-like behavior.