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Anti-addiction drug ibogaine inhibits hERG channels: a cardiac arrhythmia risk.

Xaver Koenig, Michael Kovar, Stefan Boehm, Walter Sandtner, Karlheinz Hilber

Addiction Biology March 1, 2014 DOI: 10.1111/j.1369-1600.2012.00447.x (opens in new tab)

Study at a glance

AI-extracted from the abstract
Characteristics Experimental study Peer reviewed
Population Human cells
Intervention Ibogaine
Dose therapeutic concentrations
Topics Ibogaine Addiction
Keywords Anti-addiction drug Qt interval prolongation Herg potassium channels Indole alkaloid Ibogaine safety: ibogaine Safety profile Substance Cardiotoxicity: cardiac risk Life-threatening cardiac arrhythmias
Citations 50
Key findings Therapeutic concentrations of ibogaine reduce currents through human ether-a-go-go-related gene potassium channels, providing a mechanism for potential life-threatening cardiac arrhythmias.

Abstract

Ibogaine, an alkaloid derived from the African shrub Tabernanthe iboga, has shown promising anti-addictive properties in animals. Anecdotal evidence suggests that ibogaine is also anti-addictive in humans. Thus, it alleviates drug craving and impedes relapse of drug use. Although not licensed as therapeutic drug, and despite evidence that ibogaine may disturb the rhythm of the heart, this alkaloid is currently used as an anti-addiction drug in alternative medicine. Here, we report that therapeutic concentrations of ibogaine reduce currents through human ether-a-go-go-related gene potassium channels. Thereby, we provide a mechanism by which ibogaine may generate life-threatening cardiac arrhythmias.

Comparable studies

Other experimental studies on ibogaine for addiction, most cited first.

Study Year Design Participants
Noribogaine is a G-protein biased κ-opioid receptor agonist. 2015 Experimental study with binding experiments, functional assays, and computational simulations
Anti-addiction drug ibogaine inhibits voltage-gated ionic currents: a study to assess the drug's cardiac ion channel profile. Heterologously expressed human ion channels and guinea pig cardiomyocytes 2013 Experimental study
Deconstructing the Iboga Alkaloid Skeleton: Potentiation of FGF2-induced Glial Cell Line-Derived Neurotrophic Factor Release by a Novel Compound. C6 glioma cells 2016 Laboratory study
Ibogaine signals addiction genes and methamphetamine alteration of long-term potentiation. Rat brain models 2002 Review with experimental data
Anti-addiction Drug Ibogaine Prolongs the Action Potential in Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes. Human ventricular-like cardiomyocytes derived from induced pluripotent stem cells 2017 Experimental study

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