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June 2026

Neuroplasticity

What June 2026's 15 new studies found, synthesized from the papers below. All Neuroplasticity research →

The synthesis

Synthesized from 15 studies in the library · AI-generated, grounded in the abstracts below

Found by searching the library for Neuroplasticity, neural plasticity, brain plasticity, synaptic plasticity, neurogenesis, then ranked by relevance.

Research in June 2026 found that psilocybin and ketamine can enhance neuroplasticity in animal models and humans, with psilocybin reversing stress-induced neuroplastic deficits and ketamine promoting synaptic remodeling. However, microdosing psilocin did not increase neurogenesis in rats, and the evidence for endogenous DMT maintaining neuroplasticity remains mixed. The main caveat is that most findings come from preclinical studies, with limited and preliminary human data.

Evidence by study

Direction is which way each study's own result points, not our rating of the study.

What the directions mean
Supports:
the study found the intervention worked, or its hypothesis held.
Opposes:
it found the opposite, no benefit or a harm.
No effect:
no significant difference either way.
Mixed:
effects in both directions within the same study.
Unclear:
the abstract does not report a direction.

Chronic psilocin microdosing did not affect behavior or dentate gyrus cell proliferation in rats.

preclinical RCT

Ketamine enhances synaptic plasticity and modulates neuroinflammatory pathways, offering therapeutic promise for depression and pain.

review

Psilocybin reduced fear memory and restored neuroplasticity in the hippocampus and medial prefrontal cortex in mice.

preclinical RCT

Unclear

Proposes that algorithmic art can function as a non-pharmacological neuroplastogen via prediction-error signaling.

theoretical

Evidence for endogenous DMT maintaining neuroplasticity is mixed and contested, with conflicting reports on brain concentrations.

review

Serotonergic psychedelics may enhance synaptic plasticity and modulate neuroplasticity in autism spectrum disorder.

review

Psilocybin reversed anhedonia, induced hippocampal neurogenesis, and increased BDNF expression in stressed rats.

preclinical RCT

Ketamine promotes synaptic plasticity and neurogenesis through NMDA receptor antagonism and downstream signaling.

review

AMPA receptor modulation, particularly via AMPAkines, shows antidepressant-like effects and enhances synaptic plasticity in preclinical studies.

review

Proposes a unified rescue-repair model of ketamine's anti-suicidal action involving neuroplasticity.

theoretical

Hypothesizes that psilocybin-induced neuroplasticity could theoretically modulate ADHD risk pathways, but evidence is preliminary.

theoretical

Introduces the Neural Attunement Model for post-acute stabilization of neuroplasticity after psychedelics.

theoretical

Ketamine combined with theta burst stimulation led to sustained remission and improved neuroplasticity markers (rMMN).

case report Sample size: 1

Antidepressant efficacy arises from coordinated engagement of synaptic plasticity and intrinsic excitability, with ketamine as a key example.

review

A glutamatergic augmentation strategy improved symptoms, suggesting modulation of synaptic plasticity in OCD.

case report Sample size: 1

Points of agreement

  • Psilocybin and ketamine enhance neuroplasticity in preclinical models, including synaptic remodeling, neurogenesis, and BDNF expression.
  • Glutamatergic signaling (NMDA and AMPA receptors) is a key mechanism for neuroplasticity in depression and other disorders.
  • The post-acute period after psychedelic administration may be important for stabilizing neuroplastic changes.

Conflicts

  • Psilocin microdosing did not enhance neurogenesis in rats, whereas psilocybin at higher doses did, suggesting dose-dependent effects.
  • Evidence for endogenous DMT's role in maintaining neuroplasticity is mixed, with some studies finding DMT in brain and others not.

Gaps

  • Lack of large-scale human RCTs directly measuring neuroplasticity after psychedelic or ketamine treatment.
  • Durability of neuroplastic changes beyond short-term follow-up is poorly characterized.
  • Effects in vulnerable populations (e.g., adolescents, elderly) and across different doses and regimens are understudied.
  • Standardized protocols for measuring neuroplasticity in humans are lacking.
Browse these studies in the library